To install click the Add extension button. That's it.

The source code for the WIKI 2 extension is being checked by specialists of the Mozilla Foundation, Google, and Apple. You could also do it yourself at any point in time.

How to transfigure the Wikipedia

Would you like Wikipedia to always look as professional and up-to-date? We have created a browser extension. It will enhance any encyclopedic page you visit with the magic of the WIKI 2 technology.

Try it — you can delete it anytime.

Install in 5 seconds
4,5
Kelly Slayton
Congratulations on this excellent venture… what a great idea!
Alexander Grigorievskiy
I use WIKI 2 every day and almost forgot how the original Wikipedia looks like.
Live Statistics
English Articles
Improved in 24 Hours
Added in 24 Hours
What we do. Every page goes through several hundred of perfecting techniques; in live mode. Quite the same Wikipedia. Just better.
Great Wikipedia has got greater.
.
Leo
Newton
Brights
Milds

Afferent arterioles

From Wikipedia, the free encyclopedia

Afferent arterioles
Scheme of renal tubules and its vascular supply. (Label "Afferent vessel" is visible in upper left.)
Distribution of bloodvessels in cortex of kidney.
Details
Frominterlobular artery
Suppliesglomerular capillaries
Identifiers
Latinarteriola glomerularis afferens
TA98A08.1.03.005
FMA272216 77042, 272216
Anatomical terminology

The afferent arterioles are a group of blood vessels that supply the nephrons in many excretory systems. They play an important role in the regulation of blood pressure as a part of the tubuloglomerular feedback mechanism.

The afferent arterioles branch from the renal artery, which supplies blood to the kidneys.

The afferent arterioles later diverge into the capillaries of the glomerulus.

YouTube Encyclopedic

  • 1/3
    Views:
    66 890
    28 542
    11 566
  • Glomerular Filtration: Role of Afferent and Efferent Resistance on GFR
  • Regulation of Renal Blood Flow
  • Lymphatic Vessels: Afferent & Efferent Vessels – Histology | Lecturio

Transcription

Regulation

When renal blood flow is reduced (indicating hypotension) or there is a decrease in sodium or chloride ion concentration, the macula densa of the distal tubule releases prostaglandins (mainly PGI2 and PGE2) and nitric oxide, which cause the juxtaglomerular cells lining the afferent arterioles to release renin, activating the renin–angiotensin–aldosterone system, to increase blood pressure and increase reabsorption of sodium ions into the bloodstream via aldosterone.[1]

The macula densa cell can also increase the blood pressure of the afferent arterioles by decreasing the synthesis of adenosine or ATP.[1]

If the efferent arterioles are constricted then the blood pressure in the capillaries of the kidneys will increase.

See also

Additional images

  • Malpighian corpuscle.
    Malpighian corpuscle.
  • Glomerulus.
    Glomerulus.
  • Renal corpuscle
    Renal corpuscle

References

  1. ^ a b Peti-Peterdi, János; Harris, Raymond C. (2010). "Macula Densa Sensing and Signaling Mechanisms of Renin Release: Figure 1". Journal of the American Society of Nephrology. 21 (7): 1093–1096. doi:10.1681/ASN.2009070759. PMC 4577295. PMID 20360309.

External links

This page was last edited on 4 June 2022, at 13:35
Basis of this page is in Wikipedia. Text is available under the CC BY-SA 3.0 Unported License. Non-text media are available under their specified licenses. Wikipedia® is a registered trademark of the Wikimedia Foundation, Inc. WIKI 2 is an independent company and has no affiliation with Wikimedia Foundation.
Contact WIKI 2
Introduction
Terms of Service
Privacy Policy